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It should also be noted that Finasteride can actually enhance the androgenicity of certain steroids, Nandrolone and any Nandrolone derivative such as Trenbolone being primaryexamples of this. It may be of note that there have been several trials where the effects were dose-dependant and in one experiment, it was reported that 8mg was capable of increasing testosterone levels while the 20mg dose was able to decrease them by 4.6% over the course of 20 days and that an 8mg dose also increased testosterone in rats. This reduction was noted to be independent of the dose but may just be additive and could not be the direct cause of the increase. This effect is possibly also a secondary consequence of Finasteride in that it is the enzyme Finasteride (a form of testosterone receptor substrate) that is causing the effects. This occurs because Finasteride is unable to synthesize nor does it produce the androgen receptor to the extent necessary to be capable of binding and activating (which leads to increased testosterone production). This effect should be noted against the fact that this drug is highly effective for treating androgen deficiency. It is an over-the-counter medication for male pattern baldness. In contrast to the aforementioned mechanisms by which Finasteride prevents steroidogenesis, it does not prevent this effect from occurring, and this is a major cause of its toxicity to testosterone. 5.2. Testosterone Finasteride possesses several actions that inhibit testosterone biosynthesis. The two primary actions of finasteride are decreasing the level of testosterone and decreasing the levels of 3α-hydroxysteroid dehydrogenase (SHD). Interestingly, Finasteride only has anti-androgenic activity (i.e. increasing the free testosterone to its target values) as it does not block testosterone binding to the androgen receptor. 6 Interactions with Hormones 6.1. Testosterone Dihydrotestosterone Testosterone is a well-known steroid hormone that is essential for muscle growth, and increasing levels will increase muscle mass. In response to this, a number of hormones (including estrogen of which finasteride is an inhibitor) are expressed as well as several enzymes that are regulated and also directly participate in testosterone production. Finasteride does not influence testosterone bioavailability and is thought to interfere with this process. The mechanisms of this effect are not known, yet an inhibition of the conversion of testosterone into DHT is thought to play a role and since this is not known to directly interfere with testosterone metabolism, Similar articles: